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Dr. Bryan Ardis's targeted biochemical protocol utilizing high-affinity nicotinic acetylcholine receptor (nAChR) agonists, N-Acetyl Cysteine (NAC), and buffered Vitamin C to displace neurotoxic venom-like spike peptides from alpha-7 nicotinic receptors.
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“Because the SARS-CoV-2 spike glycoprotein shares a super-antigenic toxin-like motif with high affinity for alpha-7 nicotinic acetylcholine receptors, nicotinic agonists have a thirty-fold higher binding affinity that rapidly unhooks the peptide and restores cranial nerve and vagal signaling.”
Community experiences, dosages, and questions for NAC (N-Acetyl Cysteine) in connection with Vaccine Injury
"Because the SARS-CoV-2 spike glycoprotein shares a super-antigenic toxin-like motif with high affinity for alpha-7 nicotinic acetylcholine receptors, nicotinic agonists have a thirty-fold higher binding affinity that rapidly unhooks the peptide and restores cranial nerve and vagal signaling."
The alpha-7 nicotinic acetylcholine receptor mechanism explains why cranial nerve symptoms like anosmia, tinnitus, and dysautonomia respond within days once those receptors are cleared.
Pairing the nicotinic receptor protocol with pine needle suramin tea and boron baths covers both the neurological receptor blockade and the vascular hydrogel component.
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"Because the SARS-CoV-2 spike glycoprotein shares a super-antigenic toxin-like motif with high affinity for alpha-7 nicotinic acetylcholine receptors, nicotinic agonists have a thirty-fold higher binding affinity that rapidly unhooks the peptide and restores cranial nerve and vagal signaling."